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Browse brain tumor articles via key phrases: bcnu , procarbazine , met , endpoint , symptoms; , cerebral edema , dosages , unlikely , malignant glioma , usual , somewhat , corticosteroids , administering steroids , regimens , agents , specifications , glioblastoma multiforme , 6000 rads , supratentorial malignant glioma , valid , pairwise , radiotherapy , histologically , moderately , high-dose methylprednisolone tended , protocols , related brain tumor articles: comparisons of carmustine, procarbazine, and high-dose methylprednisolone as additions to 1983 ; cancer treat rep correlations between experimental chemotherapy in the murine glioma and effectiveness of c 1978 ; cancer chemother pharmacol bcnu nsc-409962 ; and procarbazine nsc-77213 ; treatment for malignant brain tumors.
This work was supported by National Institutes of Health Grant DK 47343 to C. P. ; The costs of publication of this article were defrayed in part by the payment of page charges. This article must therefore be hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fact. To whom correspondence should be addressed: Beth Israel Deaconess Medical Center, Division of Gastroenterology, Dana 501, 330 Brookline Ave., Boston, MA 02215. Tel.: 617-667-1259; Fax: 617-667-2767; E-mail: cpothoul bidmc.harvard . 1 The abbreviations used are: SP, substance P; NK-1R, neurokinin-1 receptor; IL, interleukin; TNF , tumor necrosis factor- ; MAPK, mitogen-activated protein kinase; EGF, epidermal growth factor; EGFR.
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Ultrasonography: renal applications. J Radiol 2003; 84 12 ; : 2041-2054. 95 ; Lucidarme O, Franchi-Abella S, Correas JM, et al. Blood flow quantification with contrast-enhanced US: "entrance in the section" phenomenon-phantom and rabbit study. Radiology 2003; 228 2 ; : 298-299. 96 ; Nahar T, Li P, Kuersten B, et al. Detection of resting myocardial perfusion defects by SonoVue myocardial contrast echocardiography. Echocardiography 2003; 20 6 ; : 511-517. 97 ; Schlosser T, Veltmann C, Lohmaier S, et al. Determination of the renal blood flow in macro- and microcirculation by means of pulse inversion imaging. Fortschr Rntgenstr 2004; 176 5 ; : 724-730. 98 ; Bouakaz A, Krenning BJ, Vletter WB, et al. Contrast superharmonic imaging: a feasibility study. Ultrasound Med Biol 2003; 29 4 ; : 547-553. 99 ; Robbin ML, Lockhart ME, Barr RG. Renal imaging with ultrasound contrast: current status. Radiol Clin North 2003; 41 5 ; : 963-978. 100 ; Lefevre F, Correas JM, Briancon S, et al. Contrast-enhanced sonography of the renal transplant using triggered pulse-inversion imaging: preliminary results. Ultrasound Med Biol 2002; 28 3 ; : 303-314.
An important source of employment opportunities in the Latin American context, as in other developing economies. Though the measurement of informal activities is plagued with difficulties, several studies put the number of informal workers at between 20 and 35 percent of the urban economically active population in Latin America Portes and Schauffler [1993] ; . Measurement of informal activities in Latin America is difficult at least in part because the characterization and therefore modeling of informal markets is so complex there is no one accepted definition. Early theories considered the informal as largely a surplus labor force in urban areas, developing from high levels of migration. These workers are viewed as peripheral and having low marginal productivity. Others have characterized the informal as being not a reflection of excess labor, but of excess regulation. The informal economy is simply a mechanism for introducing market response to an overly regulated economy. A third approach proposes the informal and formal as a series of complementary networks. For a very comprehensive overview of the informal labor market concept in the Latin American context, see Portes and Schauffler [1993]. Finding ways to bring the informal sector into applied general equilibrium simulations is an important area of current research. Irrespective of the characterization of its source, the volume of unemployment and labor market structures are clearly affected by changes in other elements of the economy. Lora and Olivera [1998] comment on the role of recent macroeconomic policy. They argue that the economic reforms aimed at consolidating macroeconomic stability have been largely successful, but have resulted in significant changes in the structure of the labor market. Among the changes they identify are a greater role for services and informal employment, an increased bias towards skilled labor, and wider wage differentials. While understanding the causes and consequences of high unemployment in the Americas is difficult, it would also be remiss to ignore the issue when considering the impact of trade liberalization scenarios. On the one hand, it can be argued that trade liberalization issues and labor market policy issues are distinct.2 It is true that optimal responses will require appropriate labor market policy reform which, according to Freije [2001] has been extensive in some economies - Chile, Colombia and Peru - but not others - Venezuela and Brazil ; . However, this view ignores one of the principal virtues of AGE modeling the ability to capture the second-best implications of policy shocks. The approach that we outline below is particularly simple, consisting essentially of bounding the potential effect of trade liberalization using alternative labor market closures, but it does give us a good idea of the potential range of results and information on whether the presence of unemployment is likely to strengthen or weaken the case for liberalization. In the following section we outline the structure of our simulation model and prohibit.
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1. Harris NL, Jaffe ES, Diebold J, et al. World Health Organization classification of neoplastic diseases of the hematopoietic and lymphoid tissues: report of the clinical advisory committee meetingAirlie House, Virginia. J Clin Oncol 1999; 17: 3835 V, Propert KJ, Coleman M, et al. Alternating cycles of combination chemotherapy for patients with recurrent Hodgkin's disease following radiothereapy. A prospective randomzied study by the Cancer and Leukemia Group B. J Clin Oncol 1986; 4: 838 Hoogstraten B, Glidewell O, Holland JF, et al. Long term follow-up of combination chemotherapy-radiotherapy of stage III Hodgkin's disease. Cancer 1979; 43: 1234 DeVita VT, Jr., Serpick AA. Combination chemotherapy in the treatment of advanced Hodgkin's disease. Ann Intern Med 1970; 73: 881 DeVita VT, Jr., Simon RM, Hubbard SM, et al. Curability of advanced Hodgkin's disease with chemotherapy. Long-term follow-up of MOPP-treated patients at the National Cancer Institute. Ann Intern Med 1980; 92: 587 Stutzman L, Glidewell O. Multiple chemotherapeutic agents for Hodgkin's disease. Comparison of three routines: a cooperaetive study by Acute Leukemia Group B. JAMA 1973; 225: 396 Nissen NI, PajakTF, Glidewell O, et al. A comparative study of a BCNU containing 4-drug program versus MOPP versus 3-drug combinations in advanced Hodgkin's disease. Cancer 1979; 43: 31 Cooper MR, Pajak TF, Nissen NI, et al. A new effective four-drug combination of CCNU ; NSC-79038 ; , vinblastine, prednisone, and procarbazine for the treatment of advanced Hodgkin's disease. Cancer 1980; 46: 654 Bonadonna G, Valagussa P, Santoro A. Alternating non-cross-resistant combiantion chemotherapy or MOPP in stage IV Hodgkin's disease. Ann Intern Med 1986; 104: 739 Viviani S, Bonadonna G, Santoro A, et al. Alterneating versus hybrid MOPP and ABVD combinations in advanced Hodgkin's disease: 10-year results. J Clin Oncol 1996; 14: 1421 Canellos GP, Anderson JR, Propert KJ, et al. Chemotherapy of advanced Hodgkin's disease with MOPP, ABVD, or MOPP alternating with ABVD. N Engl J Med 1992; 327: 1478 Klimo P, Connors JM. MOPP ABV hybrid program: combination chemotherapy based on early introduction of severn effective drugs for advanced Hodgkin's disease. J Clin Oncol 1985; 3: 1174 Glick JH, Young ML, Harrington D, et al. MOPP ABV hibrid chemotherapy for advanced Hodgkin's disease significantly improves failure-free and overall survival: the 8-year results of the intergroup trial. J Clin Oncol 1998; 16: 19 Duggan DB, Petroni GR, Johnson JL, et al. Randomized comparison of ABVD and MOPP ABV hybrid for the treatment of advanced Hodgkin's disease: report of an intergroup trial. J Clin Oncol 2003; 21: 607 Dana BW, Dahlberg S, Nathwani BN, et al. Longterm follow-up of patients with low-grade malignant lymphomas treated with doxorubicin-based chemotherapy or chemoimmunotherapy. J Clin Oncol 1993; 11: 644 Kimby E, Bjorkholm M, Gahrton G, et al. Chloram bucil prednisone vs. CHOP in symptomatic low-grade non-Hodgkin's lymphomas: a randomized trial from the Lymphoma Group of Central Sweden. Ann Oncol 1994; 5: 567 McKelvey EM, Gottlieb JA, Wilson HE, et al. Hydroxyldaunomycin Adriamycin ; combination chemotherapy in malignant lymphoma. Cancer 1976; 38: 1484 Skarin AT, Canellos GP, Rosenthal DS, et al. Improved prognosis of diffuse histiocytic and undifferentiated lymphoma by use of high dose methotrexate alternating with standard agents M-BACOD ; . J Clin Oncol 1983; 1: 91 Klimo P, Connors JM. MACOP-B chemotherapy for the treatment of diffuse large-cell lymphoma. Ann Intern Med 1985; 102: 596 Longo DL, DeVitaVT, Jr., Duffey PL, et al. Superiority of ProMACE-CytaBOM over ProMACE-MOPP in the treatment of advanced diffuse aggressive lymphoma: results of a prospective randomized trial. J Clin Oncol 1991 ; 9: 25 38. Gottlieb AJ, Anderson JR, Ginsberg SJ, et al. A randomized comparison of methotrexate dose and the addition of bleomycin to CHBOP therapy for diffuse large cell lymphoma and other non-Hodgkin's lymphomas. Cancer 1990; 66: 1888 Lichtman SM, Niedzwiecki D, Barcos M, et al. Phase II study of infusional chemotherapy with doxorubicin, vincristine and etoposide plus cyclophosphamide and prednisone I-CHOPE ; in resistant diffuse aggressive non-Hodgkin's lymphoma: CALGB 9255. Ann Oncol 2000; 11: 1141 Armitage JO, Cheson BD. Interpretation of clinical trials in diffuse large-cell lymphoma. J Clin Oncol 1988; 6: 1335.
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| Procarbazine ingredientsChemotherapy is a form of targeted therapy where cytotoxic drugs act on multiplying tumor cells. The drugs can also be used as sensitizers to augment the effects of radiation therapy. Chemotherapeutic drugs can be delivered directly to brain tumors through a polymer wafer implant such as a biodegradable wafer soaked with BCNU Carmustine ; . Besides BCNU, several other chemotherapy drugs are used to treat brain tumors, which are administered by various routes. The chemotherapeutic drugs taken orally include Temozolomide TMZ, Temodar ; , procarbazine Matulane ; , and lomustine.
Table 2 ; . Among such patients, the main causes of ated including one patient who received radiation death were progressive disease and second cancers. for a relapse ; . Patients in whom MOPP-ABV treatment failure occurred or who could not be evaluated had a very poor discussion outcome Fig. 3 and Table 2 ; . The objective of this trial was to determine whether effect of pathological review radiotherapy to all initially involved areas reduces Among the 577 patients with a confirmed diagnosis the relapse rate among patients with stage III or IV of Hodgkin's lymphoma and a complete or partial Hodgkin's lymphoma who have a complete remisresponse to chemotherapy, the five-year event-free sion after MOPP-ABV chemotherapy. Our randomand overall survival rates were 80 percent and 88 ized trial involving 333 patients in complete repercent, respectively, as compared with 59 percent mission failed to show that radiotherapy increases P 0.02 ; and 65 percent P 0.008 ; , respectively, event-free or overall survival. These results underamong the 16 patients with a complete or partial re- line the effectiveness of modern chemotherapy. sponse who were found not to have Hodgkin's lym- Whereas a meta-analysis6 suggested that chemotherapy alone is sufficient treatment provided that phoma after a pathological review. eight cycles are administered, our findings indicate second cancers that six to eight cycles of MOPP-ABV produce very A second cancer developed 9 to 112 months after good results. the initiation of treatment in 6 patients in the group In the Southwest Oncology Group's randomized that did not receive radiotherapy and in 15 patients trial of involved-field radiotherapy, the 49 percent in the group that received involved-field radiother- of patients who had a complete response after six apy; the five-year cumulative rates of second can- cycles of nitrogen mustard, vincristine, prednisone, cers were 4.0 percent and 7.8 percent, respectively bleomycin, doxorubicin, and procarbazine MOP P 0.05 ; Table 2 ; . In the other groups, the five-year BAP ; were randomly assigned to receive involvedcumulative rates of second cancers approached that field radiotherapy or no further treatment.14 No among patients with a complete remission who overall benefit from radiotherapy was observed; in were assigned to receive no radiotherapy. Of the 15 patients with bulky, nodular sclerosing Hodgkin's cases of acute leukemia or myelodysplasia, which lymphoma, however, the relapse-free survival rate occurred 10 to 99 months after the initiation of was significantly higher after involved-field radiotreatment, 13 developed in patients who were irradi- therapy. A possible explanation for the difference and propylthiouracil.
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Normal results on ultrasonography of the right upper quadrant. Findings such as diffusely heterogeneous hepatic echotexture are nonspecific, representing inflammation, fatty infiltration, or fibrosis. Some investigators have found a correlation between enlarged perihepatic lymph nodes and hepatic histology 15 ; . Ultrasonographic findings of abnormal hepatic contour, enlarged portal vein, or splenomegaly suggest cirrhosis; however, cirrhosis can often be diagnosed on the basis of careful physical examination. Currently, no evidence suggests the need for routine hepatic ultrasonography in patients with a diagnosis of chronic HCV infection. In patients with established cirrhosis, screening ultrasonography may allow early identification of hepatocellular carcinoma; however, the cost-effectiveness of this practice has been questioned 16, 17 ; . Chronic HBV infection, although less common than chronic HCV infection in the United States, remains an important cause of hepatic-related illness and must be considered in patients presenting with abnormal liver chemistries. Therefore, many patients with HCV infection have already been screened for hepatitis B surface antibody and hepatitis B surface antigen during serologic evaluation. Because the modes of transmission for HBV and HCV infection overlap considerably, patients with a diagnosis of HCV should be tested for the presence of chronic HBV infection. The mode of transmission of hepatitis A virus HAV ; --the fecaloral route--differs substantially from that of HCV, and the prevalence of exposure to HAV has not been shown to be increased in patients with chronic HCV infection. However, vaccination strategies may require serologic measurement of patients' previous exposure to HAV.
| Figure 15.1. The Wave Preferences dialog box allows you to customize your view of the Analysis Window. Colors The Foreground text ; , Background, Grid and Selected Cursor colors for the Analysis Window can all be set based on the user's preference. There are also two color schemes used for the waveforms themselves, but the scheme is chosen automatically by CircuitMaker, based on the selected background color. One scheme is provided for light colored backgrounds and the other for dark colored backgrounds. Print Export Colors You have the option of printing the waveforms or exporting them to another application using the colors exactly as they appear on the screen. However, if your Analysis Window has a colored background you could end up using a lot of extra ink or toner. The second option is to print using a white background, but with colored waveforms. The third option is to print using a white background and black waveforms. If you choose the latter, you may wish to enable the Show Designation Symbols option see below and protopic.
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The above facts could induce resignation "we are the puppets of our genes and environment; we are molded by our experiences" ; , were there not the option of reworking our representation through speech. How we talk about our past, and the stories we tell ourselves about our present and our future, reflect our feelings and emotions.17 The same facts can induce different feelings because each person has a different history, and does not attribute the same emotion to the same verbal representation. The fact that the emotional response to the same story differs between listeners invites the speaker to make the effort of empathy, by which he or she looks at himself or herself from the outside. This cognitive leap can act upon and transform the initial emotion. This use of speech--this rhetoric--expresses the emotions of the inner world, organizes the behavioral consequences of these emotions, and thus explains the possibility of mental transmission. Between molecular biology on the one hand and emotion-structuring speech on the other, mood stands at a confluence of determinants, and is subject to modification by each
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