Kato 770 parts
Smallpox 80. Nakano JH. Evaluation of virological laboratory methods in smallpox diagnosis. Bull WHO. 1973; 48: 529534. Kato S, Takahashi, Kameyama, Kamahora J. A study on the morphological and cyto-immunological relationship between the inclusions of variola, cowpox, rabbitpox, vaccinia variola origin ; and vaccinia IHD and consideration of the term "Guarnieri body." Biken J. 1959; 2: 353363. Mitra AC, Sarkar SK, Mukherjee MK, Chakravarty MS. Evaluation of the precipitation-in-gel reaction in the diagnosis of smallpox. Bull WHO. 1973; 49: 555558. Dumbell KR, Bedson HS, Rossier E. The laboratory differentiation between variola major and variola minor. Bull WHO. 1961; 25: 7378. Baxby D. Jenner's Smallpox Vaccine. London, England: Heinemann Educational Books; 1981. 85. Bloch H. Edward Jenner: The history and effects of smallpox, inoculation, and vaccination. J Dis Child. 1993; 147: 772774. Baxby D. Poxviruses. In: Parker MT, Collier LH, eds. Topley and Wilson's Principles of Bacteriology, Virology and Immunity. 8th ed, vol 4. London, England: Arnold; 1990: 559571. 87. Herrlich A, Mayr A, Mahnel H, Munz E. Experimental studies on transformation of the variola virus into the vaccinia virus. Archiv fr die gesamte Virusforschung. 1963; 12: 579599. Baxby D. Vaccinia virus. In: Quinnan GV, ed. Vaccinia Viruses as Vectors for Vaccine Antigens. New York, NY: Elsevier; 1985: 1123. 89. Lane JM, Ruben FL, Neff JM, Millar JD. Complications of smallpox vaccination, 1968: Results of ten statewide surveys. J Infect Dis. 1970; 122: 303309. Centers for Disease Control. Contact spread of vaccinia from a recently vaccinated Marine--Louisiana. MMWR. 1984; 33 3 ; : 3738. 91. Centers for Disease Control. Contact spread of vaccinia from a National Guard vaccinee. MMWR. 1985; 34 13 ; : 182183. 92. Lane JM, Reuben FL, Neff JM, et al. Complications of smallpox vaccination, 1968: National surveillance in the United States. New Engl J Med. 1969; 281: 12011207. Fulginiti VA, Kempe CH, Hathaway WE, et al. Progressive vaccinia in immunologically deficient individuals. Birth Defects. 1968; 4: 129145. Freed ER, Richard JD, Escobar MR. Vaccinia necrosum and its relationship to impaired immunologic responsiveness. J Med. 1972; 52: 411420. Goldstein JA, Neff JM, Lane JM, Koplan JP. Smallpox vaccination reactions, prophylaxis and therapy of complications. Pediatrics. 1975; 55: 342347. Fenner F, Henderson DA, Arita I, Jezek Z, Ladnyi ID. Developments in vaccination and control between 1900 and 1966. In: Smallpox and Its Eradication. Geneva, Switzerland: World Health Organization; 1988: Chap 7: 302307. 97. Kempe CH. Studies on smallpox and complications of smallpox vaccination. Pediatrics. 1960; 26: 176189. Fulginiti VA, Winograd LA, Jackson M, Ellis P. Therapy of experimental vaccinal keratitis. Arch Ophthalmol. 1965; 74: 539544. Jack MD, Sorenson RW. Vaccinal keratitis treated with IDU. Arch Ophthalmol. 1963; 69: 730.
To assuage thirst from salted fish and to relieve themselves, they finally arrived at Salisbury on the next day and were marched through a cold, drizzling rain to the prison known far and wide as "one of the most loathsome in Rebeldom." Early in the war conditions were said not to have been too bad at Salisbury. Then, after a series of Rebel victories, came an influx of some 10, 000 prisoners of war. Salisbury unable adequately to handle the situation became a den of horror. Rations became scant and irregular. Communication with the outside world was cut off. Suffering for want of food became acute. Bread could be purchased for per loaf, but who had the According to Mr. Hover, his winter as an inmate at Salisbury had to be the worst of his life. Things reached a pass where men ordinarily of the highest integrity would steal, lie and cheat to get their comrades share of food. Hunger silenced the voice of conscience. Each man would draw about 13 ounces of corn bread each morning and eat it in about 13 seconds or less and then wait until the next day for more. At times, an issue of thin watery soup was given out. Many of the men had no containers for this and, to avoid the loss of such a luxury, took off one of their battered shoes to use as a soup plate. Rats and mice became delicacies, and a hapless dog that somehow wandered into the stockyard, the women's homes, in the village of Salisbury, and not one of them raised a hand to better our condition. Burial of the dead was a grisly task. Bodies would be taken to the dead house and there stripped of everything that might be of use to the living. There was no clothing issued by the prison. After the bodies had been stripped, a rough wagon drawn by four mules would be summoned and loaded with as many bodies as it would hold. This ghastly load would then be drawn to a trench dug by the prisoners and dumped therein. In such manner 11, 000 Union men died and were buried at.
Dimarzio kato group
If you answer "YES" to any of the above, please include in your proposal section B7.1 the more detailed version of Table A "Crucial information" ; obtained from: : europa .int comm research science-society ethics rules en and also incorporate in section B.7.1 and in other appropriate parts of your proposal comments corresponding to the detailed instructions given in sections C-D at the above address Table B. Proposers are requested to confirm that the proposed research does not involve: Research activity aimed at human cloning for reproductive purposes, Research activity intended to modify the genetic heritage of human beings which could make such changes heritable11 Research activity intended to create human embryos solely for the purpose of research or for the purpose of stem cell procurement, including by means of somatic cell nuclear transfer. YES NO Confirmation : the proposed research involves none of the issues listed in Table B X!
Aloyo VJ, Ruffin JS, Pazdalski PS, Kirifides AL and Harvey JA 1995 ; [3H]WIN 35, 428 binding in the caudate nucleus of the rabbit: Evidence for a single site on the dopamine transporter. J Pharmacol Exp Ther 273: 435 444. Araki T, Kato H, Shuto K, Fujiwara T and Itoyama Y 1997 ; Effect of aging on dopaminergic receptors and uptake sites in the rat brain studied by receptor autoradiography. J Neurol Sci 148: 131137. Bannon MJ and Whitty CJ 1997 ; Age-related and regional differences in dopamine transporter mRNA expression in human midbrain. Neurology 48: 969 977. Chen NH, Xu C, Coffey LL and Reith ME 1996 ; [3H]WIN 35, 428 [2 betacarbomethoxy-3 beta- 4-fluorophenyl ; tropane] binding to rat brain membranes: Comparing dopamine cell body areas with nerve terminal regions. Biochem Pharmacol 51: 563566. DeBlasi A, O'Reilly K and Motulsky HJ 1989 ; Calculating receptor number from binding experiments using same compound as radioligand and competitor. Trends Pharmacol Sci 10: 227229. Dickinson SD, Sabeti J, Larson GA, Giardina K, Rubinstein M, Kelley MA, Grandy DK, Low MA, Gerhardt GA and Zahniser NR 1999 ; Dopamine D2 receptordeficient mice exhibit decreased dopamine transporter function but no changes in dopamine release in dorsal striatum. J Neurochem 72: 148 156 Edwards RH 1993 ; Neural degeneration and the transport of neurotransmitters. Ann Neurol 34: 638 645. Fernandez-Ruiz J, De Miguel R, Hernandez ML, Cebeira M and Ramos JA 1992 ; Comparisons between brain dopaminergic neurons of juvenile and aged rats: Sex-related differences. Mechan Ageing Dev 63: 4555. Friedemann MN 1992 ; In vivo electrochemical studies of dopamine diffusion and clearance in the striatum of young and aged Fischer-344 rats. Age 15: 2328. Friedemann MN and Gerhardt GA 1992 ; Regional effects of aging on dopaminergic function in the Fischer-344 rat. Neurobiol Aging 13: 325332. Gerhardt GA, Cass WA, Henson M, Zhang Z, Ovadia A, Hoffer BJ and Gash DM 1995 ; Age-related changes in potassium-evoked overflow of dopamine in the striatum of the rhesus monkey. Neurobiol Aging 16: 939 946. Giros B, Jaber M, Jones SR, Wightman RM and Caron MG 1996 ; Hyperlocomotion and indifference to cocaine and amphetamine in mice lacking the dopamine transporter. Nature London ; 379: 606 612. Gracz LM and Madras BK 1995 ; [3H]WIN 35, 428 [3H]CFT ; binds to multiple charge-states of the solubilized dopamine transporter in primate striatum. J Pharmacol Exp Ther 273: 1224 1234. Haaparanta M, Bergman J, Laakso A, Hietala J and Solin O 1996 ; [18F]CFT [18F]WIN 35, 428 ; , a radioligand to study the dopamine transporter with PET: Biodistribution in rats. Synapse 23: 321327. Hebert MA and Gerhardt GA 1999 ; Age-related changes in the capacity, rate and.
Kato williams skate
THE CRETACEOUS-TERTIARY TRANSITION IN MEXICO AND HAITI: IMPACT AND OR VOLCANISM ? 1 ; W. STINNESBECK, P. SCHULTE, 2 ; G. KELLER, 3 ; T. ADATTE, 4 ; J.G. LOPEZ-OLIVA, 5 ; D. STBEN and U. KRAMAR. 1 ; Geologisches Institut, Universitaet Karlsruhe 76128, Germany 2 ; Department of Geosciences, Princeton University, NJ 08544, USA 3 ; Institut de Gologie, Universit de Neuchtel, 2007, Switzerland 4 ; U.A.N.L., Linares, N.L., Mexico Spherule-rich deposits SRDs ; in Mexico and Haiti are often interpreted as ejecta of the Chicxulub impact and cited in support of a K boundary age of this event. We report here on the stratigraphy, lithology and the depositional environment of over a dozen new sections which contain one or more spherule-rich deposits SRD ; in the La Sierrita area of NE Mexico and the Beloc area of Haiti. At La Sierrita, the sections reveal the presence of up to three SRDs interbedded with hemipelagic marls of latest Maastrichtian age. The marls are deposited under normal pelagic conditions with no significant evidence for reworking. Original deposition of the spherule layers is stratigraphically below siliciclastic deposits. The siliciclastic deposits contain several bioturbated horizons which indicate that deposition occurred over a long time period and was not associated with the spherule event. The K T boundary, as characterized by an Ir anomaly, is stratigraphically above the SRDs and the siliciclastic deposits in the Mexican sections. In the Beloc region, deposition of the glass spherule-rich deposit occurred within the early Danian Zone Pla. The SRD is followed by an Ir anomaly and above it a second Pd-dominated PGE anomaly. The Ir anomaly is of roughly chondritic-type and compatible with an impact event, whereas the Pddominated PGE anomaly is more compatible with an ocean flood basalt origin. This suggests a multi-event scenario consistent with one, and possibly two, impacts followed by a major volcanic event in the Caribbean.
4. Slavin S, Nagler A, Naparstek E, et al. Nonmyeloablative stem cell transplantation and cell therapy as an alternative to conventional bone marrow transplantation with lethal cytoreduction for the treatment of malignant and nonmalignant hematologic diseases. Blood 1998; 91: 756 Childs R, Chernoff A, Contentin N, et al. Regression of metastatic renal-cell carcinoma after nonmyeloablative allogeneic peripheral-blood stem-cell transplantation. N Engl J Med 2000; 343: 750 Blaise DP, Bay JO, Faucher C, et al. Reduced intensity preparative regimen and allogeneic stem cell transplantation for advanced solid tumors. Blood 2004; 103: 435 Gluckman E, Rocha V, Boyer-Chammard A, et al. Outcome of cord-blood transplantation from related and unrelated donors. Eurocord Transplant Group and the European Blood and Marrow Transplantation Group. N Engl J Med 1997; 337: 373 Rubinstein P, Carrier C, Scaradavou A, et al. Outcomes among 562 recipients of placental-blood transplants from unrelated donors. N Engl J Med 1998; 339: 156577. Laughlin MJ, Barker J, Bambach B, et al. Hematopoietic engraftment and survival in adult recipients of umbilical-cord blood from unrelated donors. N Engl J Med 2001; 344: 181522. Gardiner CM, Meara AO, Reen DJ. Differential cytotoxicity of cord blood and bone marrow-derived natural killer cells. Blood 1998; 91: 207 Rizzieri DA, Long GD, Vredenburgh JJ, et al. Successful allogeneic engraftment of mismatched unrelated cord blood following a nonmyeloablative preparative regimen. Blood 2001; 98: 3486 Wagner JE, Barker JN, DeFor TE, et al. Transplantation of unrelated donor umbilical cord blood in 102 patients with malignant and nonmalignant diseases: influence of CD34 cell dose and HLA disparity on treatment-related mortality and survival. Blood 2002; 100: 1611 Kodera Y, Morishima Y, Kato S, et al. Analysis of 500 bone marrow transplants from unrelated donors UR-BMT ; facilitated by the Japan Marrow Donor Program: confirmation of UR-BMT as a standard therapy for patients with leukemia and aplastic anemia. Bone Marrow Transplant 1999; 24: 9951003. Nishihira H, Kato K, Isoyama K, et al. The Japanese cord blood bank network experience with cord blood transplantation from unrelated donors for haematological malignancies: an evaluation of graft-versushost disease prophylaxis. Br J Haematol 2003; 120: 516 Kanda Y, Mineishi S, Saito T, et al. Long-term low-dose acyclovir against varicella-zoster virus reactivation after allogeneic hematopoietic stem cell transplantation. Bone Marrow Transplant 2001; 28: 689 Hughes WT, Armstrong D, Bodey GP, et al. 2002 guidelines for the use of antimicrobial agents in neutropenic patients with cancer. Clin Infect Dis 2002; 34: 730 Hughes WT, Armstrong D, Bodey GP, et al. 1997 guidelines for the use of antimicrobial agents in neutropenic patients with unexplained fever. Infectious Diseases Society of America. Clin Infect Dis 1997; 25: 55173. Przepiorka D, Weisdorf D, Martin P, et al. 1994 Consensus Conference on Acute GVHD Grading. Bone Marrow Transplant 1995; 15: 825 Sullivan KM, Agura E, Anasetti C, et al. Chronic graft-versus-host disease and other late complications of bone marrow transplantation. Semin Hematol 1991; 28: 250 Thiede C, Florek M, Bornhauser M, et al. Rapid quantification of mixed chimerism using multiplex amplification of short tandem repeat markers and fluorescence detection. Bone Marrow Transplant 1999; 23: 1055 Barker JN, Weisdorf DJ, DeFor TE, Blazar BR, Miller JS, Wagner JE. Rapid and complete donor chimerism in adult recipients of unrelated donor umbilical cord blood transplantation after reduced intensity conditioning. Blood. 2003; 102: 19159. Deeg HJ, Amylon ID, Harris RE, et al. Marrow transplants from unrelated donors for patients with aplastic anemia: minimum effective and kava.
Kato 550 excavator
We have previously characterized a family of modB dependent O-glycosylated proteins which are reactive with the MUD50 monoclonal antibody. One of these, the PsB glycoprotein, is part of a preassembled multiprotein complex stored in the PSVs. The PsB complex is synchronously secreted at the end of development and becomes a central part of the spore coat Watson et al., 1993, 1994 ; . In this report we have addressed two fundamental issues, viz. the biochemical function of the complex and the assembly of its protein subunits. The data show that the PsB complex binds specifically to cellulose. Moreover, the immunoprecipitation analysis of spore coat protein mutants showed that assembly of SP96 into the PsB complex is required for activity. Neither free SP96 nor a partial complex lacking SP96 show binding activity. Although the assembly of SP96 into the complex is clearly required for cellulose binding activity the precise binding site is not known. SP96 may acquire the correct conformation for binding activity once it is assembled into the complex. Alternatively, SP96 assembly into the complex may induce a conformational change in one or more of the other subunits which is required for the cellulose binding activity. Our hypothesis that the PsB complex would have this biochemical activity was based on our earlier observations that it localized to the cellulose containing spore coats Watson et al., 1994 ; . We also predicted that we would be able to demonstrate binding because the PsB complex in slug cells resides in PSVs where it is not already bound to cellulose Watson et al., 1994; West and Erdos, 1990 ; . This may help explain why no specific in vitro cellulose binding proteins have been demonstrated for plants Varner, 1989 ; . If cellulose binding proteins do exist in plants, they may be rapidly and irreversibly incorporated into the cellulose containing cell walls and would not be available for in vitro binding assays. We also have been able to demonstrate that the MUD50 reactive sheathin proteins can bind to cellulose. Earlier microscopic studies showed that these proteins co-localized with cellulose in the slug sheath as footprints where the migrating slug made contact with the substratum Zhou-Chou et al., 1995 ; . This localization suggested that these proteins may be important in slug migration and is consistent with our earlier observation that slugs of modB mutant strains cannot migrate Alexander et al., 1988 ; . In addition, at least four other Coomassie Blue staining proteins bind to cellulose. One of these bands is the discoidin proteins, but we have not yet identified any of the other three or pursued further char.
In December 1997, the Company announced that it had entered into a definitive agreement to acquire Sano Corporation "Sano" ; . This all share transaction, which was completed on 27 February 1998, values Sano at approximately US5, 000, 000. On 14 January 1998, Elan and Neuralab Limited "Neuralab" ; completed a private placement of 1, 250, 000 units. Each unit consisted of one common share of Neuralab, one initial warrant to purchase one American Depositary Share "ADS" ; of Elan at an exercise price of US.01 per ADS, and, contingent upon the occurrence of certain future events, one additional warrant to purchase one ADS at a price to be determined based on future trading prices of ADSs. The aggregate gross proceeds received by Neuralab from the sale of the units was US, 000, 000, substantially all of which will be used to reimburse Elan under a development agreement whereby Elan will conduct research and development for Neuralab to identify therapeutic compounds for use in the treatment of Alzheimer's disease and kenalog.
Kato track design
Experience With Surgical Intervention for Screening-Detected Lung Cancer Masahiro Tsuboi, Norihiko Ikeda and Harubumi Kato Chest 2004; 125; 167DOI chest.125.5 suppl.167S This information is current as of March 16, 2008.
| Naohiro kato lsuTable used to assess the weight and volumetric requirements of the AIAA cargo. Figure 2 shows the use of CADKEY to actually layout the cargo bay [10]. They determined the payload dimensions and cargo bay layout as well as the weight and center of gravity location for a variety of loading conditions. Volumetric packaging, mass properties and cg determination were well within their capability. Figure 3 shows a drawing of an entire aircraft by a freshman [11]. He actually took the lead role in the configuration layout. Examples from the Condor design team [12] include design of the cockpit layout shown in Fig. 4, while Fig. 5 illustrates the cross-section of the design for the troop cabin. The corresponding seat and emergency exit layout are given in Fig. 6. Finally, the landing gear layout is shown in Fig. 7a and 7b. These figures illustrate the many aspects of design which do not necessarily rely heavily on engineering science. The freshmen were able to proceed with consultation from team members that had taken more advanced courses. In the ozone USRA project ; group, one freshman designed the attachment system to connect a large propane tank to the airframe [13]. He said that, compared to his assignments in his engineering static's class being taken the same semester ; , this work was much more meaningful to him in learning the value of mechanics. RESULTS The results of this experiment were virtually all positive. Perhaps the best evidence of this is the fact that aircraft design competition teams with freshmen participants won first and third place in this national design contest. Three of the ten members of the third place team were freshmen and keppra.
The evaluation and clinical management of poisoned patients is challenging. Toxin exposures may be unintentionalor intentional, through environmental or occupationalexposure, as a result of therapeutic error, or malicious. Most poisonings are by ingestion. Recognizing common toxic syndromes may be critical in treating such patients. However, classic presentations of a toxic syndrome may be obscured if the ingestion includes multiple toxins. Pharmaceuticals are associated with almost three-fourths of all reported deaths and major toxicity presentations. Few specific antidotes are available for the myriad of potential poisonings. The most commonly utilized antidotes are N-acetylcysteine, naloxone, and flumazenil. Treatment in most casesinvolvesstandard decontamination procedures, monitoring, and supportivecare.The ultimatedisposition f the patient o from the emergency department may involve parental education, social work intervention, or psychiatric consultation. The role of clinical laboratorymedicine is of the utmost importance to the management of the poisoned patient. Although many immediate clinical decisions are initiated before.
Map of kato cyprus
Annual incidence of type 1 diabetes in qubec between 1989-2000 in children and ketek
| The mean yields of SCE's per chromosome in normal cells varied considerably. The range was 0.196 to 0.256. This is consistent with earlier reports on normal cells in which the mean yield of SCE's per chromosome ranged from 0.14 to 0.22 CHAGANTI, SCHONBERG GERMAN and 1974 ; , 0.20 to 0.32 LATT 1974 ; , or 0.21 to 0.40 GALLOWAY EVANS and 1975 ; . Since some of the SCE's might not be spontaneous, but induced by the BrdUrd KATO 1974a; LATT1974; WOLFF and PERRY 1974 ; , this variability might be caused by differential incorporation of BrdUrd in individual cells. In our experiments the ranges of SCE's obtained for normal, XP, galactosemic, and Lesch-Nyhan syndrome cells did not differ to any great extent Table 2 ; . If excision repair or post-replication repair were necessary for the production of SCE's, the frequencies of exchanges in repair-defective cells should be different from those in normal cells. Thus, neither the amount of excision repair in the various XP cell lines from 0% to about 50% of normal ; nor the presence of defective post-replication repair in the XP variant appear to have any effect on the yields of SCE's. This is contrary to the results obtained for chromosome aberrations, which are higher in excision-repair-defective cells than in normal cells after exposures to either UV light PARRINGTON, DELHANTY BADEN and 19 1971 ; or 4-nitroquinoline-1-oxide SASAKI 73 ; . When the results presented in Table 2 are grouped into those from untransformed cells and cells transformed by SV40 virus, there does seem to be a slightly higher yield of SCE's in the transformed cells. If this difference proves to be real, it implies that the presence of integrated viral genomes increases SCE's. None of the findings in our experiments, however, is as dramatic as the high frequency of SCE's that occurs in cells from patients with Bloom's syndrome CHAGANTI, SCHONBERG GERMAN and 1974 ; . Cells from patients with this genetic disease have 12 to 13 times more SCE's than normal. Although Bloom's syndrome is characterized by chromosome fragility and a predisposition to cancer, cells.
Supervisor Ladner discussed the semantics for payment of the Long Beach Water Management District docket. The Chancery Clerk will change the language to reflect the administrative function of the Board. c ; Supervisor Rockco inquired whether Dukes, Dukes, Keating & Faneca's fees for HCSD administrative services and civil service were paid from the Sheriff's budget. The County Administrator confirmed that that those claims were charged to the Sheriff's budget. d ; Supervisor Ladner inquired whether it was a common practice to modify and extend covenants for piece of property sold by the Harrison County Development Commission. Mr. Barnett stated that this was not a common practice and resulted from Hurricane Katrina. e ; Supervisor Martin inquired whether the Tax Collector or any other official had received a request from Winn-Dixie to reduce their tax liabilities due to terms of bankruptcy. The Board Attorney will contact Mr. Wessler for a report to the Board at the next scheduled meeting. f ; Mr. Philip Shaw who was scheduled to appear before the Board, but could not attend the meeting, will be rescheduled for September 11, 2006. g ; The Board will hold a 3rd meeting in Gulfport on September 25, 2006 09 BOARD ACTION RECESS in the First Judicial District and ketoprofen.
Kato railroading
Transmit Clock Selection: CLKXM Table 5 shows how the CLKXM bit in PCR selects the transmit clock and whether the CLKX pin is an input or output. Table 5. Transmit Clock Selection.
[69] Li R, Waga S, Hannon GJ, Beach D, Stillman B. Differential effects by the p21 CDK inhibitor on PCNA-dependent DNA replication and repair. Nature. 1994; 371 6497 ; : 534537. [70] George J, Castellazzi M, Buttin G. Prophage induction and cell division in E. coli. III. Mutations sfiA and sfiB restore division in tif and lon strains and permit the expression of mutator properties of tif. Mol Gen Genet. 1975; 140 4 ; : 309332. [71] Bi EF, Lutkenhaus J. FtsZ ring structure associated with division in Escherichia coli. Nature. 1991; 354 6349 ; : 161164. [72] Lutkenhaus JF. Coupling of DNA replication and cell division: sulB is an allele of ftsZ. J Bacteriol. 1983; 154 3 ; : 13391346. [73] Schoemaker JM, Gayda RC, Markovitz A. Regulation of cell division in Escherichia coli: SOS induction and cellular location of the sulA protein, a key to lon-associated filamentation and death. J Bacteriol. 1984; 158 2 ; : 551561. [74] Jones C, Holland IB. Role of the SulB FtsZ ; protein in division inhibition during the SOS response in Escherichia coli: FtsZ stabilizes the inhibitor SulA in maxicells. Proc Natl Acad Sci USA. 1985; 82 18 ; : 60456049. [75] Hill TM, Sharma B, Valjavec-Gratian M, Smith J. sfiindependent filamentation in Escherichia coli is lexA dependent and requires DNA damage for induction. J Bacteriol. 1997; 179 6 ; : 19311939. [76] Dorazi R, Dewar SJ. The SOS promoter dinH is essential for ftsK transcription during cell division. Microbiology. 2000; 146 11 ; : 28912899. [77] Wang L, Lutkenhaus J. FtsK is an essential cell division protein that is localized to the septum and induced as part of the SOS response. Mol Microbiol. 1998; 29 3 ; : 731740. [78] Chen JC, Beckwith J. FtsQ, FtsL and FtsI require FtsK, but not FtsN, for co-localization with FtsZ during Escherichia coli cell division. Mol Microbiol. 2001; 42 2 ; : 395413. [79] Brent R, Ptashne M. The lexA gene product represses its own promoter. Proc Natl Acad Sci USA. 1980; 77 4 ; : 19321936. [80] Yasuda T, Nagata T, Ohmori H. Multicopy suppressors of the cold-sensitive phenotype of the pcsA68 dinD68 ; mutation in Escherichia coli. J Bacteriol. 1996; 178 13 ; : 38543859. [81] Yasuda T, Morimatsu K, Kato R, Usukura J, Takahashi M, Ohmori H. Physical interactions between DinI and RecA nucleoprotein filament for the regulation of SOS mutagenesis. EMBO J. 2001; 20 5 ; : 119211202. [82] Yasuda T, Morimatsu K, Horii T, Nagata T, Ohmori H. Inhibition of Escherichia coli RecA coprotease activities by DinI. EMBO J. 1998; 17 11 ; : 32073216. [83] Ramirez BE, Voloshin ON, Camerini-Otero RD, Bax A. Solution structure of DinI provides insight into its mode of RecA inactivation. Protein Sci. 2000; 9 11 ; : 21612169 and kineret.
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T'ap pase l' nan rizib. Yo t'ap di: 42. Gade! Li sove lt moun, li pa kapab sove tt pa li. Si l' se Izrayl la, se pou l' desann sou kwa a koulye a. L sa a, n'a kw nan li. 43. Li te mete konfyans li nan Bondye, li te di se pitit Bondye li ye. Ann w koulye a si Bondye va vin delivre li. 44. Ata ansasen ki te kloure sou lt kwa yo t'ap joure l' menm jan an tou. 45. V midi konsa, vin gen yon fnwa sou tout peyi a, jouk twaz apre midi. 46. V twaz, Jezi rele byen f, li di: Eli, Eli lema sabaktani? ki vle di: Bondye, Bondye, poukisa ou lage m' konsa? ; 47. Nan moun ki te la genyen ki tande l' pale. Yo di: Men l'ap rele Eli. 48. Lamenm, yonn nan yo kouri al pran yon eponj, li tranpe l' nan veng, li mete l' nan pwent yon gl wozo, li lonje l' b bouch Jezi ba l' bw. 49. Men, lt moun yo di: Tann non, monch. Ann w si Eli ap vin delivre li. 50. Jezi bay yon gwo rl ank, epi li mouri. 51. Menm l a, rido ki te nan tanp lan chire an de moso, depi anwo jouk anba. T a tranble. Wch yo fann. 52. Tonm m yo louvri, k anpil moun pp Bondye a ki te mouri leve vivan ank, yo soti nan tonm yo. 53. Apre Jezi li menm te leve soti vivan nan lanm, yo antre lavil Jerizalm kote anpil moun te w yo. 54. L ofisye lame a ansanm ak gad ki t'ap veye Jezi ansanm av l' yo tranblemannt a ak tou sa ki te pase, yon sl lap pran yo. Yo di: Se vre wi, nonm sa a te Pitit Bondye. 55. Te gen plizy fanm la tou. Men, yo te rete lwen ap gade. Se moun ki t'ap swiv Jezi, ki t'ap okipe l' depi l l' te nan peyi Galile. 56. Nan yo te genyen: Mari, moun lavil Magdala a, Mari, manman Jak ak Jozf, ansanm ak manman pitit Zebede yo. 57. L soly fin kouche, yon nonm rich, moun lavil Arimate, vin rive. Yo te rele l' Jozf. Li menm tou li te yon disip Jezi. 58. li al jwenn Pilat, li mande l' k Jezi a. Pilat bay ld pou yo renmt li k a. 59. Jozf pran k a, li vlope l' nan yon bl dra blan tou nf. 60. Apre sa, li mete k a nan yon kavo li te fk fouye nan wch la pou tt pa l'. Apre sa, li woule yon gwo wch devan bouch kavo a. Epi li al f wout li. 61. Mari, moun lavil Magdala a, ansanm ak lt Mari a te chita la, anfas kavo a. 62. Nan denmen, ki vle di jou repo a menm, chf prt yo ansanm ak farizyen yo ale b kot Pilat. 63. Yo di li: Chf, nou chonje l nonm ki t'ap bay manti a te vivan, li te di apre twa jou li pral leve soti vivan. 64. Bay ld pou yo veye kavo a byen veye pandan twa jou. Konsa, disip li yo p'ap kapab vin pran k a pou yo di pp apre sa msye leve soti vivan nan lanm. Dnye kout manti sa a ta pase premye a. 65. Pilat di yo: Men yon eskwad gad. Ale, veye kavo a jan nou vle a. 66. Y ale. Pou yo pi asire kavo a te byen fmen, yo poze sele sou wch ki te devan bouch kavo a, epi yo mete eskwad gad la veye li and kato.
Where TI and TE are inspiratory and expiratory time s ; , respectively, and Patm is in cmH2O. The peak dT was identified, and 10 sequential breaths free from movement artifacts were measured for that period. Measurement of respiratory system impedance using the FOT. Methods for the FOT, including calibration techniques, followed previous publications 25, 54 ; . Briefly, mice were anesthetized with ketamine 50 75 mg kg ; and xylazine 5 mg kg ; Butler, Dublin, OH ; intraperitoneally, and a tracheostomy was performed with a 19-g cannula Becton Dickinson, Franklin Lakes, NY ; . Once anesthesia was confirmed by lack of response to toe pinch, mice were paralyzed with pancuronium 1 mg ml, Baxter Healthcare, Irvine, CA ; . Supplemental ketamine 25 mg kg ip ; was provided every 0.5 h. Ventilation was set at frequency of 200 breaths min, tidal volume VT ; 0.3 ml, positive end-expiratory pressure 3.0 cmH2O, and inspired oxygen was supplemented at all times. Repeated measurements were performed using a commercial data acquisition system for input impedance between 0.5 and 19.5 Hz Quick Prime 3 analyzer, FlexiVent System, SCIREQ, Montreal, Quebec ; . A constant-phase model 25 ; was employed to compute Raw "Raw-FOT" ; , in addition to tissue resistance Gti ; and elastance Hti ; coefficients. Baseline Raw, Gti, and Hti were tabulated, but the MCh responses using Gti and Hti were not used for comparisons with conscious measurements; only Raw was used for this purpose. To derive a value of sRaw i.e., Raw FRC ; from FOT for comparison with conscious sRaw-RWBP, we measured FRC using the Boyle's law method in a separate group of similarly anesthetizedtracheostomized female C57 mice n 30, 19 22 g ; using a commercial plethysmograph PLY 3111, Buxco Electronics ; , data acqui jap and klonopin.
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