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AFFIX THE BUREAU SEAL HERE 40-39-209. Except as otherwise provided in 40-39-207 a ; - d ; , no record shall be removed from the SOR, unless ordered by a court of competent jurisdiction. 40-39-210. Upon receipt of notice of the death of a registered offender, the TBI shall remove from the SOR all data pertaining to the deceased offender. 40-39-211. a ; While mandated to comply with the requirements of this chapter, no sexual offender as defined in 40-39-202 16 ; or violent sexual offender as defined in.
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Carol A. Erikson, MSPH * , Trillium, Inc., 356 Farragut Crossing Drive, Knoxville, TN 37922 After attending this presentation, attendees will learn about an unusual petroleum product fingerprint that may prove useful in determining the source or identification of released materials. This presentation will impact the forensic community and or humanity by presenting data to illustrate an unusual fingerprint in a petroleum product found in the environment; this information may offer additional forensic clues as to the source or identification of the product released. Fingerprinting techniques are commonly used in environmental forensics to identify classes of petroleum products and or to link contamination to a particular source. Gas chromatography with flame ionization detection GC FID ; , a very sensitive measurement technique for alkanes and acyclic isoprenoids e.g., pristane and phytane ; , allows evaluation of characteristic peak patterns to establish the type of petroleum product gasoline, kerosene, diesel fuel, motor oil, etc. ; present. Gas chromatography with electron capture detection GC ECD ; is not sensitive to hydrocarbons but readily detects halogenated and unsaturated materials, among others, and is often used in tandem with GC FID to evaluate the presence of additives such as the alkyl lead compounds e.g., tetraethyl lead ; . Typical GC FID and GC ECD fingerprints of a modern Jet A fuel are presented in Figure 1. The hydrocarbon pattern exhibited in the FID fingerprint is characteristic, and the ECD fingerprint gives little additional information. A recent GC FID fingerprint analysis of ground water samples taken from a petroleum storage facility showed the presence of several different petroleum products, including Jet A fuel. GC ECD fingerprints were also generated, and, as expected, most gave little additional information. In three samples, however, the GC ECD chromatogram showed a peak pattern very similar to the GC FID fingerprint but eluting approximately 8 minutes later Figure 2 ; . What was this? Too early in the chromatogram to be polychlorinated biphenyls, the ECD results were quite puzzling. To generate more information, the extract was subjected to gas chromatography mass spectrometry GC MS ; analysis to allow a closer look at these peaks, and, hopefully, to facilitate identification of the compounds they represent. The answer: a series of dialkyl disulfides, ranging from C11 to C18. ASTM specifications regarding sulfur content of Jet A fuel have not changed since 1959, when Standard Specification for Aviation Turbine Fuels ASTM D 1655 ; was first issued. So, the presence or absence of dialkyl disulfides does not appear to be a candidate for forensic age-dating based on a particular point in time when the total sulfur content of Jet A had to be reduced to meet specifications. However, high concentrations of organic sulfur compounds mercaptans ; are undesirable in petroleum refining for many reasons, including corrosivity, color changes, and unpleasant odors. Removing mercaptans from petroleum streams is a process known as sweetening. When the total sulfur content of the petroleum product is already within final product requirements, most gasoline, jet fuel, kerosene, and diesel fractions can be successfully sweetened using a caustic solution to oxidize the odorous and corrosive mercaptans to disulfides, which have much more acceptable.
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Less a group and more a collective of disparate artists united under one moniker and philosophy, Sweatshop Union is actually comprised of four groups--Dirty Circus, Pigeon Hole, Innocent Bystanders, and Kyprios--who came together nearly five years ago to pool their resources in order to more easily release an album. In doing so, Sweatshop Union's members realized that their penchant for old-school-style, give-and-take rapping over inventive beats--not to mention their desire to "say something significant" which sometimes takes the form of criticism of mainstream hip-hop ; -- was a creative currency they were only too happy to trade in. Staying true to their socially conscious ideology and collective viewpoint has proven to be a solid choice for the group, as the past few years have seen the Sweatshop Union touring with the likes of the Black Eyed Peas, Blackalicious, Jurassic 5, and many other well-known hip-hop acts who are not afraid to tip their hats to rap's fertile underground. However, when the group pays a visit at 9pm Sat., Sept. 23 to the Nightlight Lounge 211 E. Chestnut St. ; , they will be the headliners. And, if their past Bellingham shows are any indication, you're probably not going to want to miss this one. Tickets are a mere , and info can be had at nightlightlounge.
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15. Zhu H, Li Y, Trush MA. Differences in xenobiotic detoxifying activities between bone marrow stromal cells from mice and rats: implications for benzene-induced hematotoxicity. J Toxicol Environ Health 1995; 46: 183-201. Hoffmann MJ, Sinko PJ, Lee YH, Meeker RJ, Snyder R. Pharmacokinetic studies in Tg and FVB mice administered [14C] benzene either by oral gavage or intradermal injection. Toxicol Appl Pharmacol 2001; 174: 139-145. Morley A, Blake J. An animal model of chronic aplastic marrow failure. I. Late marrow failure after busulfan. Blood 1974; 44: 49-56. Botnick LE, Hannon EC, Hellman S. A long lasting proliferative defect in the hematopoietic stem cell compartment following cytotoxic agents. Int J Radiat Oncol Biol Phys 1979; 5: 1621-1625. Fitchen JH, Cline MJ. The effect of granulopoietic stress in mice with "latent" bone marrow failure. Exp Hematol 1980; 8: 788-794. Fitchen JH, Deregnaucourt J, Cline MJ. An in vitro model of hematopoietic injury in chronic hypoplastic anemia. Cell Tissue Kinet 1981; 14: 85-90. Boyd RL, Caro J, Halka KG, Erslev AJ. Granulopoiesis in long-term culture by marrow from mice with busulfan-induced chronic latent aplasia. Int J Cell Cloning 1986; 4: 357-367. Pugsley CA, Forbes IJ, Morley AA. Immunologic abnormalities in an animal model of chronic hypoplastic marrow failure induced by busulfan. Blood 1978; 51: 601-610. Ideriha NM, Vugman I, Falcao RP. Ectopic bone marrow development in experimental busulfan-induced hypoplastic anemia in mice. Blut 1984; 48: 277-284. Gibson FM, Andrews CM, Diamanti P, Rizzo S, Macharia G, Gordon-Smith EC, Williams T, Turton J. A new model of busulphan-induced chronic bone marrow aplasia in the female BALB c mouse. Int J Exp Pathol 2003; 84: 31-48. McManus PM, Weiss L. Busulfan-induced chronic bone marrow failure: changes in cortical bone, marrow stromal cells, and adherent cell colonies. Blood 1984; 64: 1036-1041. Millar JL, Hudspith BN, Blackett NM. Reduced lethality in mice receiving a combined dose of cyclophosphamide and busulphan. Br J Cancer 1975; 32: 193-198. Morley A, Trainor K, Remes J. Residual marrow damage: possible explanation for idiosyncrasy to chloramphenicol. Br J Haematol 1976; 32: 525-531. Yunis AA, Miller AM, Salem Z, Corbett MD, Arimura GK. Nitroso-chloramphenicol: possible mediator in chloramphenicol-induced aplastic anemia. J Lab Clin Med 1980; 96: 36-46. Nara N, Bessho M, Hirashima K, Momo H. Effects of chloramphenicol on hematopoietic inductive microenvironment. Exp Hematol 1982; 10: 20-25. Holt DE, Andrews CM, Payne JP, Williams TC, Turton JA. The myelotoxicity of chloramphenicol: in vitro and in vivo studies: II: In vivo myelotoxicity in the B6C3F1 mouse. Hum Exp Toxicol 1998; 17: 8-17. Turton JA, Yallop D, Andrews CM, Fagg R, York M, Williams TC. Haemotoxicity of chloramphenicol succinate in the CD-1 mouse and Wistar Hanover rat. Hum Exp Toxicol 1999; 18: 566-576. Turton JA, Havard AC, Robinson S, Holt DE, Andrews CM, Fagg R, Williams TC. An assessment of chloramphenicol and thiamphenicol in the induction of aplastic anaemia in the BALB c mouse. Food Chem Toxicol 2000; 38: 925-938. Festing MF, Diamanti P, Turton JA. Strain differences in haematological response to chloramphenicol succinate in mice: implications for toxicological research. Food Chem Toxicol 2001; 39: 375-383.
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